Aug 1, 1997. Here we report that the MAPK cascade is required for hippocampal long term potentiation (LTP), a robust and widely studied form of synaptic plasticity. We observed that PD 098059, Regulation of Extracellular Signal-Regulated Kinase by Cannabinoids in Hippocampus J. Neurosci. 2003 23: 2371-.
Regulation of extracellular signal-regulated kinase by cannabinoids. – Mar 15, 2003. Regulation of extracellular signal-regulated kinase by cannabinoids in hippocampus. Derkinderen P(1), Valjent E, Toutant M, Corvol JC, Enslen H, Ledent C, Trzaskos J, Caboche J, Girault JA. Author information: (1)Institut National de la Santé et de la Recherche Médicale/Université Pierre et Marie Curie.
E-mail: [email protected] The Raf kinases Raf-1 and B-Raf are upstream activators of the extracellular signal.
Jul 20, 2009. 2003 Regulation of extracellular signal-regulated kinase by cannabinoids in hippocampus. Journal of Neuroscience 23 2371–2382. Abstract/FREE Full Text. ↵. Devane WA,; Dysarz FA III.,; Johnson MR,; Melvin LS &; Howlett AC. 1988 Determination and characterization of a cannabinoid receptor in rat.
activate p38 mitogen-activated protein kinases through CB1 receptors in hip- pocampus. J Neurochem 77:957–960. Derkinderen P, Valjent E, Toutant M, Corvol JC, Enslen H, Ledent C, Trzaskos J, Caboche J, and Girault JA (2003) Regulation of extracellular signal-regulated kinase by cannabinoids in hippocampus.
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The nucleus accumbens (NAc) is composed of multiple neuronal. In addition, acute cocaine causes extracellular signal-regulated kinase (ERK)-dependent phosphorylation of mitogen- and stress-activated kinase 1 (MSK1; also.
The Endocannabinoids page describes the synthesis and degradation of these compounds and their role in the regulation of feeding behaviors and.
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Several studies indicated extracellular signal-regulated kinases (ERKs), the phosphatidylinositol 3-kinase with its downstream effector AKT, and the phosphatase. We propose that the endogenous cannabinoid system modulates extinction of aversive memories, at least in part via regulation of the activity of kinases and.
Introduction: alcohol is a psychotropic depressant of the central nervous system (CNS) that promotes simultaneous changes in several neuronal pathways, exerting a profound neurological impact that leads to various behavioral and biological alterations.
Regulation of Extracellular Signal-Regulated Kinase by Cannabinoids in Hippocampus. stimulation of CB1-Rs activates the extracellular-regulated kinase.
PDF Regulation of Extracellular Signal-Regulated Kinase by. – Regulation of Extracellular Signal-Regulated Kinase by Cannabinoids in Hippocampus Pascal Derkinderen,1* Emmanuel Valjent,1,2* Madeleine Toutant,1 Jean-Christophe Corvol,1 Herve´ Enslen,1
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. (THC) activated extracellular signal-regulated kinase. Regulation of Extracellular Signal-Regulated Kinase by Cannabinoids in Hippocampus.
shows that eCBs stimulate neural progenitor proliferation and inhibit hippocampal neurogenesis in normal adult brain. Cannabinoids. These new observations support that eCBs constitute a new family of lipid signaling cues responsible for the regulation of. kinase family (extracellular signal-regulated kinase (ERK),
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